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Biological Classification System

Department of Biocontainment Sciences - Revision 28.4

VITAE CLASS (Green-V)
Biohazards requiring minimal specialised containment

Vitae-class entities demonstrate biological properties that pose negligible risk to human health or ecological systems under standard laboratory conditions.Standard Protocols:
- BSL-2 equivalent facilities minimum
- Standard PPE (gloves, lab coats, eye protection)
- Routine decontamination procedures
- Weekly health monitoring for assigned personnel
- Standard biological waste disposalExamples: Benign genetic anomalies, non-pathogenic bacterial strains with unusual properties, symbiotic organisms with minimal host interaction requirements.

MORBUS CLASS (Yellow-M)
Biohazards requiring active containment measures

Morbus-class entities exhibit infectious, mutagenic, or otherwise harmful biological properties that can be reliably contained through established protocols.Standard Protocols:
- BSL-3 minimum, BSL-4 recommended
- Full hazmat suits with independent air supply
- Airlock entry/exit procedures with chemical showers
- Daily health screenings and blood work for all personnel
- Incineration of all contaminated materials
- 14-day quarantine period post-exposure
- Immediate medical intervention protocols on standbyExamples: Rapidly mutating pathogens, parasitic organisms with complex life cycles, biological agents with moderate transmission vectors.

PESTIS CLASS (Red-P)
Biohazards requiring maximum security containment

Pestis-class entities represent extreme biological threats capable of causing widespread ecological collapse, mass extinction events, or fundamental alteration of human biology.Standard Protocols:
- Custom BSL-5+ facilities with multiple redundancy systems
- Sealed environmental suits with 6-hour air supplies
- Triple airlock systems with 48-hour decontamination cycles
- Personnel require psychological evaluation and genetic screening
- All materials handled via remote manipulation only
- Site-wide lockdown procedures in case of breach
- Nuclear sterilization protocols authorized as last resort
- Minimum 500km evacuation radius for catastrophic breach scenariosExamples: Prion-based consciousness transfers, self-replicating biological weapons, organisms capable of atmospheric bioforming.

FAMES CLASS (Pink-F)
Biohazards requiring global-scale containment

Fames-class entities possess biological properties that actively resist containment and pose existential threats to human civilisation or the planetary biosphere.Standard Protocols:
- Multi-site distributed containment across isolated facilities
- Rotation of containment sites every 30 days maximum
- Personnel with no family ties or social connections preferred
- Memory modification procedures for departing staff
- Orbital monitoring stations maintain constant surveillance
- International cooperation agreements with world governments
- Contingency plans include selective population evacuation
- Species preservation protocols for worst-case scenariosExamples: Adaptive pathogens that evolve countermeasures to containment, biological entities with global-scale environmental manipulation capabilities.

VACUUM CLASS (White-V)
Biohazards requiring theoretical containment only

Vacuum-class entities are too dangerous for physical containment but must be studied through theoretical models, remote observation, or mathematical simulation.Standard Protocols:
- No direct physical interaction permitted under any circumstances
- Research conducted through quantum observation chambers only
- All data analysis performed by AI systems in isolated networks
- Human researchers limited to reviewing sanitized reports
- Facility staff unaware of entity's true nature
- Multiple facility destruction protocols on standby
- Interplanetary evacuation scenarios maintainedExamples: Biological concepts that become reality when observed, living mathematical equations that rewrite physical laws, organisms that exist in temporal loops affecting causality.

NEUTRALIZED (Gray-N)
Former biohazards rendered permanently inert

Entities that once posed biological threats but have been successfully eliminated, sterilized, or rendered permanently inactive through Foundation intervention.Standard Protocols:
- Minimal monitoring for residual biological activity
- Standard archival procedures for research data
- Periodic site inspection for environmental contamination
- Historical documentation maintained for reference purposes

PENDING (Purple-P)
Biohazards undergoing classification assessment

Newly discovered biological entities whose threat level and containment requirements are still being evaluated.Standard Protocols:
- Maximum security containment until classification complete
- Accelerated research timelines with expanded teams
- Daily threat assessment reviews
- Flexible protocol adjustments based on emerging data
- Emergency reclassification procedures available

"In biological containment, complacency breeds catastrophe. Every organism is a potential extinction event until proven otherwise."
— Dr. Princess Elena Vasquez, Director of Biocontainment Sciences

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CLASSIFIED INCIDENT REPORT

Classification Level: TOP SECRET - COMPARTMENTALIZED ACCESS ONLYDocument ID: JAI-2028-SISDYT-000Date of Compilation: [REDACTED]Reporting Agency: [REDACTED]Distribution: EYES ONLY - AUTHORIZED PERSONNEL

EXECUTIVE SUMMARY

This report documents the catastrophic containment failure and subsequent global health crisis originating from SISDYT Corporation's unauthorised pharmaceutical research program. The incident resulted in the accidental release of an experimental bioactive compound, subsequently designated as Agent Somnus-7 or Viral-Induced Anthropophagic Syndrome (VIAS), into municipal water systems across two continents.BACKGROUNDSISDYT Corporation, a multinational pharmaceutical conglomerate with defence contracts and a technological gaming company, initiated Project GARDNER in [REDACTED] with the stated objective of developing performance-enhancing stimulants for military personnel and civilian applications. The research focused on creating compounds that would extend wakefulness periods while maintaining cognitive function and physical performance.

INCIDENT TIMELINE

Phase Alpha - Initial Breach
Location: SISDYT Research Facility, Taipei, Taiwan
Date: [REDACTED]Laboratory technician [NAME REDACTED] violated established protocols by disposing of experimental compound batch SV-001 through standard drainage systems. Investigation revealed the individual lacked proper clearance and had falsified credentials during the hiring process. The compound, initially designed as a neurochemical stimulant, demonstrated unexpected mutagenic properties upon contact with environmental bacteria in the municipal water treatment system.Phase Beta - Viral Evolution
Affected Regions: Greater Taipei Metropolitan AreaInitial exposure manifested through municipal tap water consumption. Subjects exhibited increased metabolic rates and enhanced appetite without corresponding weight gain. The compound demonstrated rapid adaptation, evolving from a simple stimulant into a self-replicating viral agent designated Agent Somnus-7.Transmission vectors expanded beyond water consumption to include:
- Direct fluid exchange (saliva, blood, semen, vaginal fluids)
- Sexual contact
- Open wound contaminationPhase Gamma - Pharmaceutical Acceleration
Timeline: 14-21 days post-initial exposureCivilian medical interventions inadvertently accelerated viral evolution. Standard pharmaceuticals—including antibiotics, antivirals, and over-the-counter medications—provided selective pressure that enhanced the pathogen's resistance mechanisms and virulence. The virus demonstrated unprecedented adaptive capabilities, developing immunity to conventional treatments within 72-hour exposure cycles.Phase Delta - Secondary Release
Location: SISDYT Subsidiary Laboratory, Houston, Texas, USA
Date: [REDACTED]The same compromised technician, having relocated to the United States facility, repeated the disposal violation using a concentrated version of the compound (batch SV-007). This release introduced a significantly more potent variant into the North American water grid.

CURRENT STATUS

Taiwan Response
Taiwanese authorities implemented immediate water system shutdown and established quarantine protocols. Infected individuals were successfully isolated for research purposes.
Current containment status: STABLE.United States Response
Federal response was delayed due to inter-agency coordination failures. Infected individuals breached containment and crossed into neighboring territories before effective barriers were established.International Containment MeasuresCanadian Border: Military-enforced quarantine zone established along the 49th parallel. Armed checkpoints with shoot-to-kill authorization for unauthorized crossing attempts.
Current classification: HOSTILE CONTAINMENT.Mexican Border: Enhanced barrier construction completed. Strict prohibition on entry for any individuals with United States citizenship or recent travel history.
Current classification: SELECTIVE CONTAINMENT.

THREAT ASSESSMENT

Agent Somnus-7 demonstrates characteristics of both viral and prion-based pathogens. Long-term effects remain under investigation, but preliminary data suggests irreversible neurological modifications in advanced-stage subjects.Recommended Classification: KETER-CLASS BIOLOGICAL HAZARD[END REPORT]This document contains information affecting the national security of [REDACTED]. Unauthorized disclosure is punishable under Section [REDACTED] of the Espionage Act.

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Pathophysiological Analysis of Viral-Induced Anthropophagic Syndrome (VIAS): A Comprehensive Study of Infection Mechanisms and Host Behavioral Modifications

Abstract
This report presents a detailed analysis of a viral pathogen responsible for inducing severe behavioral and physiological alterations in human hosts, collectively termed Viral-Induced Anthropophagic Syndrome (VIAS). The causative agent demonstrates unique tropism for adipose tissue and exhibits complex neurotropic properties resulting in profound alterations to feeding behavior, metabolic processes, and post-mortem host manipulation.Introduction
The emergence of VIAS represents a significant departure from conventional viral pathogenesis models. Unlike typical neurotropic viruses that primarily target neural tissue, this pathogen demonstrates a multi-system approach, initiating infection through adipocyte manipulation before progressing to neurological invasion. The syndrome's most distinctive characteristic involves the development of compulsive anthropophagic behavior coupled with metabolic dysregulation that ultimately proves fatal to the host while paradoxically sustaining viral replication.Methods
Viral transmission was observed and analyzed in controlled laboratory settings using infected human tissue cultures and rodent models to trace viral replication and metabolic disruptions. Histological analyses were conducted on adipose tissues to observe leptin levels, and enzymatic assays were performed to measure amylase activity in saliva samples from infected subjects.

Results

Transmission and Initial Infection Pathways
The viral agent demonstrates broad transmission capabilities through multiple bodily fluid vectors, including hemolymph, saliva, cerebrospinal fluid, and contaminated aqueous solutions. Upon entering the host organism, the virus exhibits selective tropism for white adipose tissue (WAT), specifically targeting mature adipocytes within subcutaneous and visceral fat deposits.The initial cellular invasion mechanism involves viral attachment to leptin receptors present on adipocyte cell surfaces. Following endocytosis, the virus hijacks the host cell's leptin production machinery, resulting in aberrant overexpression of this satiety hormone. Paradoxically, despite elevated circulating leptin concentrations that should theoretically suppress appetite, infected individuals experience progressive hyperphagia due to concurrent neurological modifications.Metabolic Alterations and Digestive System Modifications
Viral infection triggers significant alterations in salivary gland function, particularly affecting the parotid and submandibular glands. Infected acinar cells begin producing abnormally high concentrations of α-amylase, the primary enzyme responsible for starch hydrolysis. Concurrently, sucrase-isomaltase expression increases dramatically along the intestinal brush border, resulting in accelerated disaccharide breakdown.This enzymatic upregulation creates a hyperefficient carbohydrate processing system, rapidly depleting available glucose stores and triggering persistent gluconeogenic stress responses. The metabolic imbalance contributes to the characteristic ravenous hunger observed in infected individuals, as the body's energy demands exceed normal dietary intake capabilities.Neurological Pathogenesis and Behavioral Modifications
The virus demonstrates remarkable neurotropic capabilities, utilizing retrograde axonal transport to migrate from peripheral infection sites to central nervous system targets. Primary viral invasion occurs through the trigeminal and facial nerve pathways, which provide direct connections between salivary glands and brainstem nuclei controlling feeding behavior.Upon reaching the hypothalamus, the virus specifically targets the arcuate nucleus and lateral hypothalamic area, regions critical for appetite regulation and feeding drive. Viral replication within these areas disrupts normal satiety signaling pathways, creating a pathological state of persistent hunger despite adequate or excessive caloric intake.The progression from normal feeding behavior to anthropophagic compulsion occurs through systematic destruction of higher cortical inhibitory controls. As the virus spreads to prefrontal cortical regions responsible for impulse control and social cognition, infected individuals lose the ability to distinguish between appropriate and inappropriate food sources. The detection of elevated body temperature in potential prey triggers overwhelming predatory responses, likely mediated through alterations in thermosensitive neurons within the preoptic area.Metabolic Incompatibility and Host Deterioration
Human digestive physiology lacks the specialized adaptations necessary for processing raw mammalian tissue as a primary nutritional source. Unlike obligate carnivores, humans possess limited pepsin production capacity and lack the robust stomach acid concentrations required for efficient protein denaturation and pathogen neutralization in uncooked meat.The consumption of large quantities of raw human tissue introduces numerous pathogenic organisms, including bacteria, parasites, and prions, overwhelming the compromised immune system of infected individuals. Additionally, the absence of specialized hepatic enzymes for processing certain amino acid profiles found in muscle tissue leads to toxic metabolite accumulation.The combination of nutritional deficiencies, pathogen overload, and metabolic toxicity results in rapid systemic organ failure. Hepatic necrosis, renal dysfunction, and cardiovascular collapse typically occur within 72-96 hours of initial anthropophagic behavior onset.Post-Mortem Viral Persistence and Host Reanimation
Following clinical death, the virus faces an existential crisis as cellular metabolism ceases and traditional replication mechanisms become unavailable. However, the pathogen has evolved a remarkable survival strategy involving post-mortem host manipulation.Viral particles concentrated within the brainstem and spinal cord maintain minimal cellular functions through anaerobic metabolic pathways. By preserving basic motor neuron function and maintaining rudimentary cardiovascular circulation, the virus sustains sufficient tissue oxygenation to prevent complete cellular death in critical neural regions.The reanimated host continues exhibiting feeding behaviors, though now driven purely by viral survival needs rather than biological hunger. The virus requires constant protein intake to maintain minimal cellular repair mechanisms and prevent complete tissue degradation. This creates the characteristic "undead" state where the host appears deceased but continues hunting behavior.Termination Protocols and Viral Elimination
Given the virus's concentration within central nervous system tissues and its ability to maintain post-mortem neural function, complete viral elimination requires destruction of the brainstem and upper cervical spinal cord. Cranial trauma sufficient to disrupt the medulla oblongata and upper cervical segments effectively terminates viral function by eliminating the minimal life support systems maintaining the reanimated state.Alternatively, complete cervical decapitation achieves the same result by severing the neural pathways necessary for maintaining basic motor functions and circulatory support. Without these minimal physiological processes, the virus cannot sustain cellular integrity and undergoes rapid degradation.

Discussion

This study elucidates the complex interaction between a neuro-pathogenic virus and its host, leading to societal threats through zombification. The rapid progression from infection to aggressive behavior underscores the need for immediate response mechanisms in potential outbreak scenarios. Further research is needed to explore potential antiviral treatments that could inhibit leptin manipulation or amylase enhancement, providing avenues for medical intervention.Conclusion
VIAS represents a unique viral pathogen with unprecedented host manipulation capabilities extending beyond clinical death. The syndrome's progression from metabolic dysregulation to behavioral modification to post-mortem reanimation demonstrates remarkable viral adaptability and survival strategies. Understanding these mechanisms provides crucial insights for developing targeted therapeutic interventions and containment protocols for future outbreak scenarios.Further research should focus on identifying specific viral proteins responsible for adipocyte targeting and post-mortem neural preservation, as these represent potential therapeutic targets for preventing infection progression or achieving viral elimination in infected hosts.

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The Story

Just a few years before the second Red Scare, in 1944, the US and USSR were developing stronger soldiers. The Axis Powers and the Allies were already producing drugs and studying nutrition/fitness, but the USSR had the idea of combining animals with humans.Genetics in the 1940s was rudimentary, and they lacked the understanding of DNA, cells, and proteins. Additionally, heredity was still being researched. However, there was enough understanding that if you put sperm and an egg cell together, a baby would be.Through (unethical) trial and error, the first Transgenic Human(that lived in adulthood) was created. It was a Snow Leopard Transgenic Human(and they didn’t know this before, but there was only a 15% difference in the DNA of Snow Leopards and Humans).The Snow Leopard, simply known as Снежок, became an excellent soldier known for its impressive scent and prowess on the battlefield. Снежок was made to breed with a human woman(and a snow leopard), but its sperm was infertile.The US took an interest in Снежок when they had met it, but were barred from knowing how to recreate one. A CIA agent had smuggled in some copies of lab reports, and with the help of SISDYT, they were able to successfully recreate a Bloodhound Transgenic Human.For most of the years, the topic of Demihumans(Transgenic Humans) was locked down tight(in America, at least). None of the civilians knew that America was working on making TGs. Not until a (secret)journalist stumbled on this information in Argentina from her drunken husband during her vacation. She posted it anonymously in South America, and it became a hot topic. Later, the US was forced to come clean. Luckily, the (privileged) citizens didn’t feel betrayed.By 1951, the US and the USSR had successfully created a company(unit) of Demihumans. US propaganda portrayed it so that only Demihumans would fight, meaning fewer Americans would die on the battlefield.
USSR propaganda was made so that the citizens would want to become demihumans, promoting the benefits of becoming a half-human, half-animal. Considering it was patriotic.In 1957, the need to have an army wasn’t as strong, and Transgenic humans became citizens. However, they were treated as lower-class citizens.